CALGARY, CANADA--Researchers at the University of Calgary
Medical School have presented the visual evidence of Mercury's effect on
growing nerve cells, another correlation to Alzheimer's disease. These
findings may influence the future acceptance of mercury-containing
vaccines and "mercury/silver amalgam" dental fillings.
Utilizing digital time-lapse photography to display the damage to nerve
cells that minute amounts of mercury cause, researchers Leong, Syed and
Lorscheider experimented with mercury levels that are similar to or below
those levels of mercury found in the brains of humans with "mercury/silver
amalgam" dental fillings.
The peer-reviewed study published in the prestigious journal NeuroReports provides visual documentation of the biochemical
mechanism by which the introduction of mercury induces hallmark diagnostic
markers indistinguishable from those seen in the Alzheimer's diseased
brain. The authors note that, to date, no other material or metal tested,
including aluminum, has produced even remotely similar reactions.
The broadcast quality video and animation documenting the biochemical
process of mercury on the nerve cells is available to interested members
of the press through Miss Karen Thomas, Media Relations, University of
Calgary, Faculty of Medicine T: 403-2202945 F: 403-210-8141 Email: Thomas@ucalgary The video can be
viewed using Quicktime 4.1 at http://commons.ucalgary.ca/mercury .
The video that accompanies the study entitled, "Retrograde Degeneration of
Neurite Membrane Structural Integrity of Nerve Growth Cones Following In
Vitro Exposure to Mercury" was funded by the International Academy of Oral
Medicine and Toxicology (IAOMT), the recognized stakeholder experts on
Mercury in Canada.
The IAOMT was formed to review, support, and disseminate research on the
suitability of materials and methodologies used in the dental practice.
The IAOMT has funded previous research by Dr. Murray Vimy on the mercury
vapors released from mercury amalgam fillings during and after chewing,
animal research showing pathopysiological damage to sheep and monkeys from
dental amalgam mercury vapor exposure. Collaborative research with the
Calgary authors of this current study and Dr. Boyd Haley at the University
of Kentucky demonstrated Alzheimer's disease-like brain damage to rats
from inhaled mercury vapor.
Dr. Haley, commenting on the importance of this new documentation, "Seven
of the characteristic markers that we look for to distinguish Alzheimer's
disease can be produced in normal brain tissues, or cultures of neurons,
by the addition of extremely low levels of mercury*. In addition, research
has shown that Alzheimer's diseased patients have at least 3 times higher
blood levels of mercury than controls. How much more research is necessary
before the appropriate regulatory bodies respond with restrictions on the
use of mercury-leaking dental amalgam fillings?"
*Alzheimer's diseased brain has been shown to contain dysfunctional
tubulin, creatine kinase and glutamine synthetase, hyperphosphorylated tau,
low levels of reduced glutathione, elevated amyloid protein and
neurofibrillar tangles.
This study confirms the
role that dental mercury from amalgam fillings plays in the development of
Alzheimer's Disease (AD). Although the American, Canadian, and Alberta
Dental Association would like to have you believe otherwise, science has
clearly demonstrated that there is a positive correlation between brain
mercury levels and the number and surfaces of "mercury/silver" amalgam
dental fillings. The mercury levels that caused the devastating damage to
nerve cells in the above referenced study were 100 to 1000 times those
found in the brains of people with "mercury/silver" amalgam dental
fillings.
In 1997, researchers at the University of Calgary Medical School and
the College of Pharmacy at the University of Kentucky clearly demonstrated
that exposing rats to the same levels of mercury vapor that can be
released from "mercury/silver" amalgam dental fillings caused the mercury
to interact with brain tubulin and disassemble microtubles that maintain
neurite structure. The identical neurochemical lesion of similar or
greater magnitude is evident in Alzheimer brain homogenates from
approximately 80% of patients, when compared to human age-matched
neurological controls. (Neurotoxicology 1997;18(2):315-324)
In 2000, researchers at the Neurobiology Laboratory, Psychiatric
University Hospital in Basel, Switzerland using neuroblastoma cells
exposed to mercury demonstrated an increase in production of amyloid
protein that makes up the amyloid plaques as well as significantly
increasing the phosphorylation of Tau protein. (J Neurochem 2000
Jan;74(1):231- 236)
Studies demonstrating a correlation between amalgam dental fillings and
brain mercury levels: Lakartidningen 1986 Feb 12;83(7):519-522
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